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A major defect in cross-regulation involves defective function of regulatory T cells (Treg), often associated with gene variants such as FOXP3 and IL2RA that prevent suppression of this autoimmune cascade [59,60,61,62]

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112 In particular, controlling IGF-1 signal transduction is a critical component underlying the antitumor effects of CR

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PD is a multifaceted disorder involving a complex interplay of biochemical and molecular mechanisms that contribute to its progression